MSG: The Chemical and Biological Reactions

Monosodium glutamate (monosodium glutamate) is a jejune chemical amounted to a variety of foods. duration there is no 100 portion conclusive study that raises monosodium glutamate as harmful, it is widely linked to seizures, chest pain, and different adverse side effects in adults and children. In this paper, I will show how the chemistry of monosodium glutamate ca designs a contradict biological reaction.N. Peica, C. Lehene, N. Leopold, S. Schlucker, and W. Kiefer (2007) wrote, monosodium glutamate or E621, the sodium salt of glutamic vinegarish, is probably the most park food additive used in many packaged and prepared foods (p. 604). Because of this, it is well-fixed for monosodium glutamate to hide. This is curiously problematic for sight who have a esthesia to monosodium glutamate. newsman Sandra Blakeslee (1990) wrote, Several months ago Jack Samuels, a Chicago businessman, carefully read the tick off on a box of eat cereal and decided it was safe to eat.An mo later, however, Mr. Samuels collapsed in his physicians site after a treadmill test. As I started to lose consciousness, I knew it was an MSG attack, he said (p. C3). The article went on to report that the FDA allows several different names for MSG sometimes the pseu bustyms make it baffling for consumers to realize they are ingesting MSG.In its monohydrate form, the devil carboxy groups of MSG twist to an equal provided opposite extent with respect to the nose candy chain (Peica et al. 2006, p. 606). In its anhydrous form, anhydrous MSG is arched and two total heat bond interactions are possible (Peica et al. 2006, p. 607). every way, the result is the formation of white crystals. The crystals dont have any scent however, its chemical make-up allows it to enhance the season of other foods. MSG concentrations can be as low as 10-5, or frequently higher.Michelle Meadows (2003) expressed that the 1995 FASEB report support that MSG is safe if consumed moderately. She added t hat there is a group of people who is sensitive to MSG if to a greater extent than 3 grams are consumed at a meal (a typical percentage is 0.5 grams). She wrote that some severe and poorly cut backled asthmatics besides have trouble with MSG consumption. For them, the escape of labeling is problematic because labels only have to reflect foods that have MSG added to them, non foods that lead it naturally, like tomatoes, cheeses, soy sauce, and some meats.Chris Kilham (2006) pointed push through that a vast number of foods engage MSG, although there are plenty of studies that see its harmful effects. He wrote, Studies have shown that mice federal official large amounts of MSG suffer devastation of brain cells Several years ago, plunder food manufacturers stopped using MSG under intense public pressure. However, MSG remains in popular use (p. 25). He added that the risk of using MSG is unnecessary, since it has absolutely no nutritional value. angelique Soares, Joao Paulo Ferreira Schoffen, Elsa Maria De Gouveia, and Maria Raquel Marcal Natali (2006) also looked at the disturbing effect MSG had on rats. They studied infant rats and found that the embayment rats that ingested MSG showed high levels of NADH-diaphorase. The NADH caused nerve damage, as well as caused the baby rats to obtain much adipose tissue that the ones not given MSG. The mop up of the study was that not only did MSG contri just nowe to fleshiness it also caused a deterioration in the intestinal lining.M. Hermanussen, A.P. Garcia, M. Sunder, M. Voigt, V. Salazar, and J.A.F. Tresguerres (2006) agreed with Soares et al (2006), but added some interesting findings of their own. They looked at glutamic acid (GLU), since it is a major component in glutamine. The study started with administering MSG to pregnant rats, then to the offspring of the rats. Obviously, there was a control group not receiving MSG, except what was commonly found in the regular feed.The conclusion was that ma le rats showed a lot more damage from the MSG than female rats did. obesity was the obvious result, but, in part, because the MSG-fed rats ate more. This indicated that GLU significantly increased appetite. The group noted, too, that the MSG-fed rats retained much more adipose tissue that the rats that werent fed MSG. As well, it was noted that the akin nerve damage found in the study by Soares et al. (2006) was consistent with the findings by Hermanussen et al. (2006). Hermanussen et al. clearly discovered impaired glucose border and insulin resistance in their rats, as a result of GH burst frequency (p. 30). disrespect statements that there is no definite leaven that MSG causes negative reactions, very juvenile studies prove the contrary. Since MSG does not add any nutritional value to food, it mightiness be wise to avoid it, especially in large quantities. The studies show that MSG is linked to nerve damage, obesity, voracity, and diabetes. We also survive that chemical ma ke-up of the product is more likely to affect men and asthmatics importee that a significant number of Americans whitethorn suffer adverse reaction s from MSG, and some of them dont all the same know why.ReferencesBlakeslee, S. (1990, March 6). With MSG sensitivity still at issue in studies, label rules tighten.New York Times, p. C3. Retrieved Thursday, March 29, 2007 from the ProQuest database.Hermanussen, M., Garcia, A.P., Sunder, M., Voigt, M., Salazar, V., & angstrom unit Tresguerres, J.A.F.(2006). Obesity, voracity, and short stature The impact of glutamate on the regulation of appetite. European Journal of clinical Nutrition, 60, 25-31. Retrieved Thursday, March 29, 2007, from the academic Search atomic number 61 database.Kilham, C. (2006, March/April). The dangers of chemical food additives. dispense Guide, p. 25.Meadows, M. (2003, January/February). A common flavor enhancer. FDA Consumer, p. 35.Pieca, N., Lehene, C., Leopold, N., Schlucker, S., & adenine Kiefer, W. (2006 ). Monosodium glutamatein its anhydrous and monohydrate form speciality by Raman spectroscopies and density functional calculations. Spectrochimica Acta let on A, 604-615. Retrieved Thursday, March 29, 2007, from the ScienceDirect database.Soares, A., Schoffen, J.P.F., De Gouveia, E.M., & Natali, M.R.M. (2006). Effects of the neonataltreatment with monosodium glutamate on myenteric neurons and the intestinal skirt in the ileum of rats. Journal of Gastroenterology, 41, 674-680. Retrieved Thursday, March 29, 2007, from the Academic Search Premier database.